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Wat (wie) is cytostatic$18639$ - definitie

DRUG THERAPY AGAINST CANCER OR INFECTIONS
Cancer Chemotherapy; Cytotoxic drug; Side effects of chemotherapy; Antineoplastic; Chemotheraputic; Chemotherapeutic; Cytostatics; Antitumour antibiotic; Chemotherapies; Cytotoxic antibiotics; Chemotherapeutics; Chemo therapy; Antineoplastic agents; Chemotherapy, cancer, regional perfusion; Anti-tumor; Cancer Drug; Anticancer agents; Chemotherapy agent; Anti-tumor compound; Anticancer antibiotic; Anti-proliferative; Antimitotic drug; Anti-mitotic; Antineoplastic antibiotic; Antitumor antibiotic; Antineoplastic agent; Cytotoxic drugs; Antineoplastic drug; Chemotherapy for cancer; Antineoplastic chemotherapy; Anti-neoplastic chemotherapy; Chemothreapy; Oral chemotherapy; Antitumor drug; Chemopreventive; Chemotherapeutic agents; Anticancer drugs; Antineoplastic medicine; Chemotherapy (oncology); Cancer drug; Cancer chemotherapy; Anti-cancer drugs; Anti-cancer drug; Antitumour; Khemotherapy; Chemotherapeutic drugs; Chemotherapy treatment; Antimitotic agents; Cancer drugs; Cancer Drugs; Chemotherapy for animals; Side effects of cancer treatment; Anticancer drug; Non-cancer chemotherapy; Antineoplastic drugs; Chemotherapeutic drug; High-dose alkylating chemotherapy; Palliative chemotherapy; Anticancer agent; Chemotherapy drug; Tumoricidal; Chemical therapy; Chemotherapy (cancer treatment); Maintenance chemotherapy; Chemotherapy medication; Cytotoxic antibiotic; Topical chemotherapy; Cytostatic antineoplastic agent; Anti-neoplastic; Adverse effects of chemotherapy; Anti-cancer medication; Anti-tumour agent; Anti-tumor agent; Anti-tumor agents; Anti-tumour agents; Antitumour agents; Antitumour agent; Antitumor agent; Antitumor agents; Anticancer medication; Chemo
  • The four phases of the cell cycle. G1 – the initial growth phase. S – the phase in which DNA is synthesised. G2 – the second growth phase in preparation for cell division. M – mitosis; where the cell divides to produce two daughter cells that continue the cell cycle.
  • Dose response relationship of cell killing by chemotherapeutic drugs on normal and cancer cells. At high doses the percentage of normal and cancer cells killed is very similar. For this reason, doses are chosen where anti-tumour activity exceeds normal cell death.<ref name=Corrie />
  •  Two DNA bases that are cross-linked by a nitrogen mustard. Different nitrogen mustards will have different chemical groups (R). The nitrogen mustards most commonly alkylate the N7 nitrogen of guanine (as shown here) but other atoms can be alkylated.<ref name =Siddik />
  • [[Deoxycytidine]] (left) and two anti-metabolite drugs (center and right), [[gemcitabine]] and [[decitabine]]. The drugs are very similar but they have subtle differences in their [[chemical structure]].
  • 5-FU dose management results in significantly better response and survival rates versus BSA dosing.<ref name="individual fluorouracil" />
  • [[Scanning electron micrograph]] of [[mesoporous silica]], a type of [[nanoparticle]] used in the delivery of chemotherapeutic drugs
  • ''Vinca'' alkaloids prevent the assembly of microtubules, whereas taxanes prevent their disassembly. Both mechanisms cause defective mitosis.
  • Two girls with [[acute lymphoblastic leukemia]] receiving chemotherapy. The girl on the left has a [[central venous catheter]] inserted in her neck. The girl on the right has a [[peripheral venous catheter]]. The arm board stabilizes the arm during needle insertion. Anti-cancer IV drip is seen at top right.
  • [[Sidney Farber]] did pioneering work in chemotherapy.
  • Topoisomerase I and II Inhibitors
  • 5-FU dose management avoids serious side effects experienced with BSA dosing.<ref name="individual fluorouracil" />

Estromustine         
CHEMICAL COMPOUND
Estrone-cytostatic complex; Leo 271 f; Leo 271 F; LEO 271 F; LEO 271 f; Estrone cytostatic complex
Estromustine (developmental code name Leo 271 f), also known as estrone 17β-3-N-bis(2-chloroethyl)carbamate or estrone–cytostatic complex, is a major active metabolite of the cystotatic antineoplastic agent and estrogen estramustine phosphate, a medication used in the treatment of prostate cancer.
Cytostasis         
Cytostatic; Antiproliferative; Antiproliferative agent; Anti-proliferative agent; Cystotatic; Cytostatic agent; Cytostatic drug
Cytostasis (cyto – cell; stasis – stoppage) is the inhibition of cell growth and multiplication. Cytostatic refers to a cellular component or medicine that inhibits cell division.
List of chemotherapeutic agents         
DRUGS THAT ARE USEFUL AGAINST CANCER
Chemotherapeutic agent
This is a list of chemotherapeutic agents, also known as cytotoxic agents or cytostatic drugs, that are known to be of use in chemotherapy for cancer. This list is organized by type of agent, although the subsections are not necessarily definitive and are subject to revision.

Wikipedia

Chemotherapy

Chemotherapy (often abbreviated to chemo and sometimes CTX or CTx) is a type of cancer treatment that uses one or more anti-cancer drugs (chemotherapeutic agents or alkylating agents) as part of a standardized chemotherapy regimen. Chemotherapy may be given with a curative intent (which almost always involves combinations of drugs) or it may aim to prolong life or to reduce symptoms (palliative chemotherapy). Chemotherapy is one of the major categories of the medical discipline specifically devoted to pharmacotherapy for cancer, which is called medical oncology.

The term chemotherapy has come to connote non-specific usage of intracellular poisons to inhibit mitosis (cell division) or induce DNA damage, which is why inhibition of DNA repair can augment chemotherapy. The connotation of the word chemotherapy excludes more selective agents that block extracellular signals (signal transduction). The development of therapies with specific molecular or genetic targets, which inhibit growth-promoting signals from classic endocrine hormones (primarily estrogens for breast cancer and androgens for prostate cancer) are now called hormonal therapies. By contrast, other inhibitions of growth-signals like those associated with receptor tyrosine kinases are referred to as targeted therapy.

Importantly, the use of drugs (whether chemotherapy, hormonal therapy or targeted therapy) constitutes systemic therapy for cancer in that they are introduced into the blood stream and are therefore in principle able to address cancer at any anatomic location in the body. Systemic therapy is often used in conjunction with other modalities that constitute local therapy (i.e., treatments whose efficacy is confined to the anatomic area where they are applied) for cancer such as radiation therapy, surgery or hyperthermia therapy.

Traditional chemotherapeutic agents are cytotoxic by means of interfering with cell division (mitosis) but cancer cells vary widely in their susceptibility to these agents. To a large extent, chemotherapy can be thought of as a way to damage or stress cells, which may then lead to cell death if apoptosis is initiated. Many of the side effects of chemotherapy can be traced to damage to normal cells that divide rapidly and are thus sensitive to anti-mitotic drugs: cells in the bone marrow, digestive tract and hair follicles. This results in the most common side-effects of chemotherapy: myelosuppression (decreased production of blood cells, hence also immunosuppression), mucositis (inflammation of the lining of the digestive tract), and alopecia (hair loss). Because of the effect on immune cells (especially lymphocytes), chemotherapy drugs often find use in a host of diseases that result from harmful overactivity of the immune system against self (so-called autoimmunity). These include rheumatoid arthritis, systemic lupus erythematosus, multiple sclerosis, vasculitis and many others.